TAILIEUCHUNG - Traffic air pollution and mortality from cardiovascular disease and all causes: a Danish cohort study

The Swiss work was based on a provisional national PM10 emission inventory. It was first disaggregated to a km2 grid. Dispersion functions for primary PM10 emission were defined in an empirical dispersion model which was used to calculate the concentration of primary PM10. The contribution of secondary particles was modelled by using simple relationships between precursor and particle concentration. The long-range transported fraction was taken from European scale models. The PM10 fractions were then summed to create the PM10 map. The traffic related part was modelled separately, using both the road-traffic related portion of PM10 emission and the respective portion of the precursor emission for secondary. | Raaschou-Nielsen et al. Environmental Health 2012 11 60 http content 11 1Z60 ENVIRONMENTAL HEALTH RESEARCH Open Access Traffic air pollution and mortality from cardiovascular disease and all causes a Danish cohort study Ole Raaschou-Nielsen1 Zorana Jovanovic Andersen1 2 Steen Solvang Jensen3 Matthias Ketzel3 Mette Sorensen1 Johnni Hansen1 Steffen Loft4 Anne Tjonneland1 and Kim Overvad5 Abstract Background Traffic air pollution has been linked to cardiovascular mortality which might be due to co-exposure to road traffic noise. Further personal and lifestyle characteristics might modify any association. Methods We followed up 52 061 participants in a Danish cohort for mortality in the nationwide Register of Causes of Death from enrollment in 1993-1997 through 2009 and traced their residential addresses from 1971 onwards in the Central Population Registry. We used dispersion-modelled concentration of nitrogen dioxide NO2 since 1971 as indicator of traffic air pollution and used Cox regression models to estimate mortality rate ratios MRRs with adjustment for potential confounders. Results Mean levels of NO2 at the residence since 1971 were significantly associated with mortality from cardiovascular disease MRR 95 confidence interval CI per doubling of NO2 concentration and all causes MRR 95 CI per doubling of NO2 concentration after adjustment for potential confounders. For participants who ate 200 g of fruit and vegetables per day the MRR was 95 CI for mortality from cardiovascular disease and 95 CI for mortality from all causes. Conclusions Traffic air pollution is associated with mortality from cardiovascular diseases and all causes after adjustment for traffic noise. The association was strongest for people with a low fruit and vegetable intake. Keywords Traffic Air pollution Cardiovascular mortality Total mortality Cohort Background Although several recent studies have shown associations

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