TAILIEUCHUNG - Critical Care Obstetrics part 28

Critical Care Obstetrics part 28 provides expert clinical guidance throughout on how you can maximize the chances of your patient and her baby surviving trauma. In this stimulating text, internationally recognized experts guide you through the most challenging situations you as an obstetrician are likely to face, enabling you to skillfully:Recognize conditions early-on which might prove life threatening, Implement immediate life-saving treatments in emergency situations, Maximize the survival prospects of both the mother and her fetus | Cardiac Disease some cases an additional 50 by the late second stage. The potential for further dramatic volume shifts occurs around the time of delivery both secondary to postpartum hemorrhage and as the result of an autotransfusion occurring with release of vena caval obstruction and sustained uterine contraction. Such volume shifts may be poorly tolerated by women whose cardiac output is highly dependent on adequate preload pulmonary hypertension or in those with fixed cardiac output mitral stenosis . Figure illustrates the marked fluctuations in cardiac output associated with normal labor delivery and postpartum 17 . The risk classification presented in Table assumes clean delineation of various cardiovascular lesions. Unfortunately in Table Coagulation factors and inhibitors during normal pregnancy. Factor Non-pregnant Late pregnancy Factor I fibrinogen 200-450mg dL 400-650mg dL Factor II prothrombin 75-125 100-125 Factor V 75-125 100-150 Factor VII 75-125 1 50-250 Factor VIII 75-150 200-500 Factor IX 75-125 100-150 Factor X 75-125 1 50-250 Factor XI 5-125 50-100 Factor XII 75-125 100-200 Factor XIII 75-125 35-75 Antithrombin III 85-110 75-100 Antifactor Xa 85-110 75-100 Reprinted by permission from Hathaway WE Bonnar J. Coagulation in pregnancy. In Hathaway WE Bonnar J eds. Perinatal Coagulation. New York Grune Stratton 1978. actual practice this is only rarely the case. Optimal management of a patient with any specific combination of lesions requires a thorough assessment of the anatomic and functional capacity of the heart followed by an analysis of how the physiologic changes described previously will impact on the specific anatomic or physiologic limitations imposed by the intrinsic disease. Such an analysis will allow a prioritization of often conflicting physiologic demands and greatly assist the clinician in avoiding or managing potential complications. Certain management principles generally apply to most patients with cardiac disease.

TỪ KHÓA LIÊN QUAN
TAILIEUCHUNG - Chia sẻ tài liệu không giới hạn
Địa chỉ : 444 Hoang Hoa Tham, Hanoi, Viet Nam
Website : tailieuchung.com
Email : tailieuchung20@gmail.com
Tailieuchung.com là thư viện tài liệu trực tuyến, nơi chia sẽ trao đổi hàng triệu tài liệu như luận văn đồ án, sách, giáo trình, đề thi.
Chúng tôi không chịu trách nhiệm liên quan đến các vấn đề bản quyền nội dung tài liệu được thành viên tự nguyện đăng tải lên, nếu phát hiện thấy tài liệu xấu hoặc tài liệu có bản quyền xin hãy email cho chúng tôi.
Đã phát hiện trình chặn quảng cáo AdBlock
Trang web này phụ thuộc vào doanh thu từ số lần hiển thị quảng cáo để tồn tại. Vui lòng tắt trình chặn quảng cáo của bạn hoặc tạm dừng tính năng chặn quảng cáo cho trang web này.