TAILIEUCHUNG - Clinical hemodynamics - Textbook (Second edition): Part 2

(BQ) Continued part 1, part 2 of the document Clinical hemodynamics - Textbook (Second edition) has contents: Right-sided heart disorders, pulmonary hypertension and related disorders, pericardial disease and restrictive myocardial diseases, left-ventricular hemodynamics, heart failure, and shock, congenital heart disease, and other contents. Invite you to refer. | RIGHT-SIDED HEART DISORDERS Michael Ragosta CHAPTER 7 After Werner Forssmann boldly inserted a urological catheter into his own right atrium the right heart became accessible to clinical investigation allowing the study of right-heart physiology in both normal and diseased The right heart is affected by many cardiac disease states. Recall that the prii left- cause of right-heart failure is left-heart failure therefore the myriad cardiac disorders associated with heart failure syndromes often impact right-heart hemodynamics. In addition numerous congenital heart conditions as well as disorders of the pericardium affect right-heart hemodynamics. The influences these conditions have on right-heart hemodynamics are discussed in their respective chapters. This chapter will focus on disorders unique to the right heart including tricuspid and pulmonic valvular diseases and the hemodynamics of right-ventricular failure with a focus on right-ventricular infarction. TRICUSPID VALVE STENOSIS This rare valvular lesion is most often due to rheumatic heart disease and is almost always associated with mitral stenosis isolated rheumatic tricuspid stenosis is very Only occasionally is tricuspid stenosis caused by other conditions including carcinoid syndrome endomyocardial fibrosis congenital tricuspid valve stenosis endocarditis pacemaker lead-related leaflet fibrosis or atrial myxoma. In the current era perhaps the most commonly observed cause of tricuspid stenosis is dysfunction of a prosthetic tricuspid valve. Tricuspid stenosis impairs right-atrial emptying and elevates right-atrial pressure. Diminished filling of the right ventricle reduces cardiac output. In cases of rheumatic heart disease the combination of tricuspid and mitral stenosis reduces the cardiac output to levels lower than expected on the basis of either valvular lesion alone. Clinical consequences of severe tricuspid stenosis include fatigue caused by low cardiac output elevated jugular veins .

• Y khoa - Dược
• Clinical hemodynamics
• Right sided heart disorders
• Pulmonary hypertension
• Left ventricular hemodynamics
• Heart failure
• Congenital heart disease
• Hemodynamic assessment
• H ypertrophic cardiomyopathy
• Related conditions
• Aortic valve disease
• Advances in hemodynamic research
• Clinical application of hemodynamic research
• Circulatory physiology
• Hemodynamics in coronary arterial disease
• Myocardial perfusion
• Application to cardiovascular surgery
• BMC Anesthesiology
• Portal pressure
• Blood loss
• Postoperative renal functions
• Systemic hemodynamics
• Pediatric cardiology
• Diagnose cardiac conditions in children
• Diagnose cardiac conditions
• Physiology of congenital heart disease in children
• Physiology of congenital heart disease
• Classification congenital heart disease in children
• Classification congenital heart disease
• Echocardiography in congenital heart disease
• Echocardiography in pediatric
• Miscellaneous cardiovascular lesions
• Anomalies of ventricular myocardium
• Acquired pediatric heart disease
• BMC Pediatrics
• Ischemic disease
• Unhealthy lifestyles
• Introduction to cardiac ultrasound imaging
• Quantitative methods
• Comprehensive surgical management
• Congenital heart surgery
• Pediatric cardiac intensive care
• Cardiac transplantation
• Myocardial protection
• Surgical technique
• Moss & adams heart disease
• Congenital cardiovascular malformations
• diseases of the endocardium
• Pulmonary vascular disease
• The young adult
• Other special problems
• Unusual forms of congenital heart disease
• Unique cardiac conditions
• Congestive heart failure in infants and children
• Increased risk
• Risk factors associated
• Multiple heart defects
• Delayed corrective surgery
• MCQs for cardiology knowledge based assessment
• Cardiology knowledge based assessment
• Ischaemic heart disease
• Valvular heart disease
• Adult congenital heart disease
• Pathophysiology of heart diseas
• The cardiomyopathies
• Mechanisms of cardiac arrhythmias
• Clinical aspects of cardiac arrhythmias
• Diseases of the pericardium
• Atrial septal defect
• Ventricular septal defect
• Complete atrioventricular canal
• Truncus arteriosus
• Infant care
• Bronchiolitis hospitalization
• Hospital discharge
• Low cardiac output syndrome
• Risk factor
• Cardiac surgery
• Left ventricular ejection fraction
• NKX2 5
• Somatic mutation
• Single nucleotide polymorphisms
• Formalin fixed
• Paraffin embedded
• Ultra fast anesthesia
• Low weight children
• Extubation time
• endothelial progenitor cells
• pulmonary arterial hypertension
• Atrial aeptal defect
• Critical congenital heart disease
• Clinically non significant CHDs
• Echocardiographic screening
• Screening methods
• Predictive markers
• Cardiac function and fetal brain MR
• Transposition of great arteries
• Near infrared spectroscopy
• Newborn infants
• Cardiac arrest
• Cardiopulmonary resuscitation
• Chromosome microarray analysis
• Copy number variation
• Single Nucleotide Polymorphism arrays
• BMC Genomics
• DNA methylation
• Monozygotic twins
• Common complex diseases
• Ultrasound assisted technique
• Conventional puncture method
• The saphenous vein
• medical research
• therapeutic
• pharmaceutical
• Abnormal Brain
• Newborns
• Respiratory syncytial virus
• Respiratory tract infections
• Palivizumab prophylaxis
• The EACVI Echo handbook
• Heart valve disease
• Right heart function
• Pulmonary artery pressure
• Critically ill patients
• Diseases of the aorta
• Stress echocardiography