TAILIEUCHUNG - MECHANISMS IN PARKINSON’S DISEASE – MODELS AND TREATMENTS

The mechanisms accounting for poor expression of the BCR in CLL remain elusive. There is no evidence of genetic defects in the BCR components [62, 63] and in contrast with their poor expression at the membrane level, transcription and intra-cellular synthesis of BCR components are normal [63, 64]. However, they cannot be assembled and transported from the endoplasmic reticulum to the cell surface because of a folding and glycosolation defect of the mu and CD79a chains though not of the CD79b chain. The poor expression of the CD22 molecule in B-CLL cells, was also found to result as a consequence. | MECHANISMS IN PARKINSON S DISEASE - MODELS AND TREATMENTS Edited by Juliana Dushanova Mechanisms in Parkinson s Disease - Models and Treatments Edited by Juliana Dushanova Published by InTech Janeza Trdine 9 51000 Rijeka Croatia Copyright 2012 InTech All chapters are Open Access distributed under the Creative Commons Attribution license which allows users to download copy and build upon published articles even for commercial purposes as long as the author and publisher are properly credited which ensures maximum dissemination and a wider impact of our publications. After this work has been published by InTech authors have the right to republish it in whole or part in any publication of which they are the author and to make other personal use of the work. Any republication referencing or personal use of the work must explicitly identify the original source. As for readers this license allows users to download copy and build upon published chapters even for commercial purposes as long as the author and publisher are properly credited which ensures maximum dissemination and a wider impact of our publications. Notice Statements and opinions expressed in the chapters are these of the individual contributors and not necessarily those of the editors or publisher. No responsibility is accepted for the accuracy of information contained in the published chapters. The publisher assumes no responsibility for any damage or injury to persons or property arising out of the use of any materials instructions methods or ideas contained in the book. Publishing Process Manager Silvia Vlase Technical Editor Teodora Smiljanic Cover Designer InTech Design Team First published February 2012 Printed in Croatia A free online edition of this book is available at Additional hard copies can be obtained from orders@ Mechanisms in Parkinson s Disease - Models and Treatments Edited by Juliana Dushanova p. cm. ISBN 978-953-307-876-2 INTECH open .

• Y học thường thức
• Chronic Inflammation
• Gastroenteropancreatic Neuroendocrine
• Chromogranin A
• Neuroendocrine Tumors
• Circulating Markers
• Gastroenteropancreatic Neuroendocrine
• BMC Cancer
• Pancreatic cancer
• Three specific biomarkers
• Cox proportional multivariable hazards
• BMC Pharmacology and Toxicology
• Phosphatidylserine containing liposomes
• Reduce inflammation
• Pegylated PSL
• Genome Biology
• NSAID treatment
• Cancer prevention
• Hallmarks of cancer
• CXC chemokine ligand 16
• End stage renal disease
• Chronic inflammation plays
• Purinergic receptor P2X ligand gated ion channel 7
• Monocyte chemotactic protein 1
• Oesophageal cancer
• Several human cancers
• Viral infections
• Tumour related inflammation
• Chronic irritation
• BMC Musculoskeletal Disorders
• Interleukin 6
• Intermuscular adipose tissue
• Quadricep muscles
• BMC Pulmonary Medicine
• Chronic inflammatory disease
• Asthmatic airway inflammation
• Neurogenic airway inflammation
• Airway secretion
• Bronchial circulation
• Rho kinase
• Asthma model
• Lung mechanics
• Guinea pigs
• Chronic airway inflammation
• Physical exercise
• Asthma therapy
• Systematic review
• Meta analysis
• Systemic inflammation
• Pulmonary function
• Chronic spinal cord injury
• Inflammatory markers
• Randomized control trials
• Sebaceous gland hyperplasia
• Diagnostic trial
• Chronic inflammatory responses
• Environmental triggers
• Bacterial infection
• Chronic inflammation of airways
• Acute exacerbations
• Streptococcus pneumoniae
• Chronic obstructive pulmonary disease
• Neutrophilic airway inflammation
• Trimethoprim sulfamethoxazole
• Magnesium isoglycyrrhizinate
• Inflammatory factors IL 6
• Histopathological analysis
• Chronic pain
• Temporomandibular joint dysfunction syndrome
• Chronic inflammation of muscle
• Tumor–stroma interactions
• Chronic pancreatitis
• Surgical PDAC specimens
• Disease specific survival
• BMC Geriatrics
• Chronic venous leg ulcers
• Social genomics
• Psychosocial stressors
• Particularly loneliness
• Chronic hepatitis C
• Sustained virological response
• Hepatocellular carcinoma
• Histological fibrosis
• Persistent histological inflammation
• Allergic asthma
• Interleukin 22
• Animal model
• Ovalbumin sensitization
• Chromogranin
• Molecular Determinants
• High Fertility
• Systems Biology
• Tight junction
• Colorectal cancer
• Chronic ethanol feeding
• Dextran sulfate sodium
• Junction protein expression
• Disease progression
• Lung function
• Airway remodeling
• Cigarette smoke
• BMC Molecular and Cell Biology
• Wound healing
• Platelet rich plasma
• Skin equivalent
• Type2 Diabetes Mellitus