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Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành hóa học dành cho các bạn yêu hóa học tham khảo đề tài: Effect of anti-inflammatory agents on transforming growth factor beta over-expressing mouse brains: a model revised | Journal of Neuroinflammation BioMed Central Research Open Access Effect of anti-inflammatory agents on transforming growth factor beta over-expressing mouse brains a model revised Pierre Lacombe1 2 Paul M Mathews3 Stephen D Schmidt3 Tilo Breidert4 Michael T Heneka5 Gary E Landreth6 Douglas L Feinstein7 and Elena Galea 1 7 Address 1Laboratoire de Recherches Cérébrovasculaires CNRS FRE 2363 Paris France 2Génétique des Maladies Cérébrovasculaires INSERM E365 Paris France 3Center for Dementia Research Nathan Kline Institute New York University School of Medicine Orangeburg New York U.S.A 4Neurologie Expérimentale et Thérapeutique INSERM U289 Hôpital de la Pitié-Salpêtrière Paris France 5Department of Neurology University of Bonn Germany 6Department of Neurosciences Alzheimer Research Laboratory Case Western Reserve University School of Medicine Cleveland Ohio U.S.A and 7Department of Anesthesiology College of Medicine University of Illinois at Chicago U.S.A Email Pierre Lacombe - lacombe@ext.jussieu.fr Paul M Mathews - mathews@nki.rfmh.org Stephen D Schmidt - Schmidt@nki.rfmh Tilo Breidert - tilo.breidert@gmx.de Michael T Heneka - Michael.Heneka@ukb.uni-bonn.de Gary E Landreth - gel2@po.cwru.edu Douglas L Feinstein - dlfeins@uic.edu Elena Galea - Elena.Galea@uab.es Corresponding author Published 02 July 2004 Received 02 June 2004 Journal of Neuroinflammation 2004 1 11 doi 10.1186 1742-2094-1-11 Accepted 02 July 2004 This article is available from http www.jneuroinflammation.cOm content 1 1 1 1 2004 Lacombe et al licensee BioMed Central Ltd. This is an Open Access article verbatim copying and redistribution of this article are permitted in all media for any purpose provided this notice is preserved along with the article s original URL. Abstract Background The over-expression of transforming growth factor P-1 TGF-P1 has been reported to cause hydrocephalus glia activation and vascular amyloidp AP deposition in mouse brains. Since these phenomena partially mimic the .
