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Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care giúp cho các bạn có thêm kiến thức về ngành y học đề tài: A quantitative analysis of the acidosis of cardiac arrest: a prospective observational study. | Available online http ccforum.eom content 9 4 R357 Research A quantitative analysis of the acidosis of cardiac arrest a prospective observational study Jun Makino1 Shigehiko Uchino2 Hiroshi Morimatsu3 and Rinaldo Bellomo4 1Staff specialist in emergency Tertiary Emergency Medical Center Tokyo Metropolitan Bokuto Hospital Tokyo Japan 2Staff specialist in intensive care Department of Emergency and Critical Care Medicine Saitama Medical Center Saitama Medical School Saitama Japan 3Staff specialist in intensive care Department of Anesthesiology and Resuscitology Okayama University Medical School Okayama Japan 4Director of intensive care research Department of Intensive Care and Department of Medicine Austin Repatriation Medical Centre Melbourne Australia Corresponding author Jun Makino makinet@jt7.so-net.ne.jp Received 11 Jan 2005 Revisions requested 22 Feb 2005 Revisions received 27 Mar 2005 Accepted 25 Apr 2005 Published 23 May 2005 Critical Care 2005 9 R357-R362 DOI 10.11 86 cc3714 This article is online at http ccforum.com content 9 4 R357 2005 Makino et al. licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Open Access Abstract Introduction Metabolic acidosis is common in patients with cardiac arrest and is conventionally considered to be essentially due to hyperlactatemia. However hyperlactatemia alone fails to explain the cause of metabolic acidosis. Recently the Stewart-Figge methodology has been found to be useful in explaining and quantifying acid-base changes in various clinical situations. This novel quantitative methodology might also provide useful insight into the factors responsible for the acidosis of cardiac arrest. We proposed that hyperlactatemia is not the sole cause of cardiac arrest acidosis and that other .
