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Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Cellular and molecular mechanisms of cigarette smoke-induced lung damage and prevention by vitamin C. | Journal of Inflammation BioMed Central Open Access Cellular and molecular mechanisms of cigarette smoke-induced lung damage and prevention by vitamin C Shuvojit Banerjee1 Ranajoy Chattopadhyay2 Arunava Ghosh1 Hemanta Koley3 Koustubh Panda1 Siddhartha Roy4 Dhrubajyoti Chattopadhyay1 and Indu B Chatterjee 1 Address 1Dr. B. C. Guha Centre for Genetic Engineering and Biotechnology University College of Science Kolkata 700019 India 2Sealy Center for Molecular Science University of Texas Medical Branch Galveston Texas 77555-1079 USA 3National Institute of Cholera and Enteric Diseases P33 CIT Road Kolkata 700010 India and 4Indian Institute of Chemical Biology 4 Raja S. C. Mullick Road Kolkata 700032 India Email Shuvojit Banerjee - sb76@rediffmail.com Ranajoy Chattopadhyay-ran72@rediffmail.com Arunava Ghosh - arunavaghosh6@yahoo.co.in Hemanta Koley-hemantakoley@hotmail.com Koustubh Panda - pandak66@yahoo.co.uk Siddhartha Roy - siddhartharoy@iicb.res.in Dhrubajyoti Chattopadhyay-d_jc@sify.com Indu B Chatterjee - ibc123@rediffmail.com Corresponding author Published II November 2008 Received II March 2008 Accepted 1 1 November 2008 Journal of Inflammation 2008 5 21 doi 10.1186 1476-9255-5-21 This article is available from http www.journal-inflammation.com content 5 1 21 2008 Banerjee et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http creativecommons.org licenses by 2.0 which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Cigarette smoke-induced cellular and molecular mechanisms of lung injury are not clear. Cigarette smoke is a complex mixture containing long-lived radicals including p-benzosemiquinone that causes oxidative damage. Earlier we had reported that oxidative protein damage is an initial event in smoke-induced lung injury. Considering that p-benzosemiquinone may be a causative factor of .
