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Tóm tắt tỷ lệ mắc bệnh thận mãn tính đang gia tăng, và các mô hình của Loạn dưỡng xương do thận dường như được chuyển từ trình bày hyperparathyroid cổ điển với một doanh thu xương thấp. Bệnh nhân bị bệnh dai dẳng cũng sống lâu hơn so với trước đây và hoạt động thể chất hơn. | Renal Osteodystrophy Nirmal C. Tejwani MD Aaron K. Schachter MD Igor Immerman BS Pramod Achan MBBS FRCS Orth Abstract The incidence of chronic renal disease is increasing and the pattern of renal osteodystrophy seems to be shifting from the classic hyperparathyroid presentation to one of low bone turnover. Patients with persistent disease also live longer than previously and are more physically active. Thus patients may experience trauma as a direct result of increased physical activity in a setting of weakened pathologic bone. Patient quality of life is primarily limited by musculoskeletal problems such as bone pain muscle weakness growth retardation and skeletal deformity. Chronic renal disease also increases the risk of comorbidity such as infection bleeding and anesthesia-related problems. Current treatment strategies include dietary changes plate-and-screw fixation and open reduction and internal fixation. Dr. Tejwani is Associate Professor Department of Orthopaedics Bellevue Hospital NYU-Hospital for Joint Diseases New York NY. Dr. Schachter is Resident NYU-Hospital for Joint Diseases. Mr. Immerman is a Medical Student NYU-Hospital for Joint Diseases. Dr. Achan is Fellow Department of Orthopaedics NYU-Hospital for Joint Diseases. None of the following authors or the departments with which they are affiliated has received anything of value from or owns stock in a commercial company or institution related directly or indirectly to the subject of this article Dr. Tejwani Dr. Schachter Mr. Immerman and Dr. Achan. Reprint requests Dr. Tejwani Bellevue Hospital 550 First Avenue NBV 21W37 New York NY 10016. J Am Acad Orthop Surg 2006 14 303-311 Copyright 2006 by the American Academy of Orthopaedic Surgeons. Renal osteodystrophy refers to pathologic bone conditions in patients with known kidney disease. The kidneys monitor the physiologic homeostasis of mineral metabolism thus any deficiency in their operation directly affects bone mineralization because of the .
