Đang chuẩn bị liên kết để tải về tài liệu:
CCL5-CCR5 interactions modulate metabolic events during tumor onset to promote tumorigenesis

In earlier studies we have shown that CCL5 activation of CCR5 induces the proliferation and survival of breast cancer cells in a mechanistic target of rapamycin (mTOR)-dependent manner and that this is in part due to CCR5-mediated increases in glycolytic metabolism. | CCL5-CCR5 interactions modulate metabolic events during tumor onset to promote tumorigenesis