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The morbidity of sepsis induced acute kidney injury remains unacceptable high and the mechanisms of that disease remains unclear. For urine backleak and intercellular tight junction among tubular epithelial cells (TECs) destruction often occur during sepsis induced acute kidney injury, we examined whether lipopolysaccharide could damage intercellular tight junction among TECs and associated mechanisms in our present study. | Lipopolysaccharide mediates the destruction of intercellular tight junction among renal tubular epithelial cells via RhoT1/SMAD-4/JAM-3 pathway
