TAILIEUCHUNG - Báo cáo y học: "Green tea increases anti-inflammatory tristetraprolin and decreases pro-inflammatory tumor necrosis factor mRNA levels in rats"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Green tea increases anti-inflammatory tristetraprolin and decreases pro-inflammatory tumor necrosis factor mRNA levels in rats. | Journal of Inflammation BioMed Central Research Open Access Green tea increases anti-inflammatory tristetraprolin and decreases pro-inflammatory tumor necrosis factor mRNA levels in rats Heping Cao 1 Meghan A Kelly1 Frank Kari2 Harry D Dawson1 Joseph F Urban Jr1 Sara Coves3 Anne M Roussel4 and Richard A Anderson 1 Address 1Nutrient Requirements and Functions Laboratory Beltsville Human Nutrition Research Center Agricultural Research Service US Department of Agriculture Building 307C BARC-East 10300 Baltimore Avenue Beltsville MD 20705 USA 2Office of Clinical Research National Institute of Environmental Health Sciences National Institutes of Health Department of Health and Human Services Research Triangle Park NC 27709 USA 3Unilever France F92842 Rueil Malmaison France and 4Laboratoire de NVMC Nutrition Vieillissement et Maladies Cardiovasculaires Faculte de Pharmacie Joseph Fourier University Domaine de la Merci 38700 La Tronche France Email Heping Cao - Meghan A Kelly - Frank Kari - kari1@ Harry D Dawson - Joseph F Urban - Sara Coves - Anne M Roussel - Richard A Anderson - Corresponding authors Published 05 January 2007 Received 20 October 2006 Accepted 05 January 2007 Journal of Inflammation 2007 4 1 doi 1476-9255-4-1 This article is available from http content 4 1 1 2007 Cao et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Background Tristetraprolin TTP ZFP36 family proteins have anti-inflammatory activity by binding to and destabilizing pro-inflammatory mRNAs such as Tnf mRNA

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